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Fernando Cabieses, Raúl Jerí and Rodolfo Landa

, definite evidence of recent tissue damage was found surrounding the vessels. c) A few, scattered, small arteries of the peduncular and caudal thalamic regions showed degenerative alterations of the endothelium. d) In the vicinity of the blood vessels of the mesencephalic and caudal diencephalic regions, there was globular swelling, rarefaction of tissue, and loss of the outline and degenerative changes of the myelin sheaths, which was much more striking and generalized at the level of both corticopontine tracts in the peduncle ( Fig. 13 ) Fig. 13

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Richard C. Schneider and Elizabeth C. Crosby

motor areas of the cerebral cortex. In this case when the discharges from the posterior lobe of the cerebellum to higher levels were unregulated largely by the efferent cerebral cortex centers again a handicapping imbalance had been set up. Removal of considerable portions of the posterior lobe of the cerebellum lessened the cerebellar discharge to midbrain and thalamic regions (and so indirectly to cerebral cortex) and thus established a smoother motor performance, with more satisfactory tonus, although the brain lesions had been increased. The various other surgical

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Alfred J. Luessenhop, William A. Shevlin, A. A. Ferrero, David C. McCullough and Bartolo M. Barone

history. This depended chiefly on the rate of development of an abnormal state of consciousness and lateralizing sensory or motor symptoms. Hemorrhages originating in the capsular or thalamic regions usually showed both flaccid hemiplegia and alteration of consciousness. In lateral capsular hemorrhages the motor symptoms developed more slowly, followed by impairment of consciousness. Hemorrhages extending into the frontal, occipital, or temporal lobes were more apt to have intense headache, followed by alteration of consciousness and less severe lateralized motor signs

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Richard P. Greenberg and Thomas B. Ducker

eighth nerve and cochlear nucleus, either absent or abnormal, and a loss of subsequent longer-latency ABEP waves. Furthermore, they reported that brain-stem gliomas were associated with a greater than 90% prevalence of ABEP abnormalities as they often infiltrate widely prior to clinical decompensation. These authors and others have correctly localized intracranial tumors (such as germinoma of the pineal area, metastatic carcinoma, brain-stem meningioma, acoustic neuroma) to the peripheral, pontomedullary, pontine, midbrain, and thalamic regions by studying changes in

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Gideon Findler, Moshe Feinsod, Graciela Lijovetzky and Moshe Hadani

corpus callosum & bilateral thalamic regions Shuping, et al. , 1980 60 glioma lt posterior thalamic region Findler, et al. , 1983 67 metastasis of bladder carcinoma rt posterior thalamic region Various explanations have been suggested as to the etiology of TGA in patients with brain tumors; these include convulsive phenomena, 4, 12 occlusion of a blood vessel by the tumor mass, 9 and hemorrhage within the tumor with subsequent sudden expansion. 17 However, a normal EEG, and the lack of hemorrhage or midline shift on the CT

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Donn M. Turner, John C. Vangilder, Saeid Mojtahedi and Eric W. Pierson

month later. Fig. 8. Case 3. Computerized tomography scan displaying an area of hemorrhage near the left midbrain and left hypothalamic-thalamic regions. Discussion Since the first clinical description of “pulsating exophthalmos” by Travers in 1809, 41 CCF has been a well studied and frequently reported entity. 1–7, 9–13, 15–17, 19–22, 24, 26–30, 33–35, 38–41, 43, 44, 46 Its incidence is estimated to be one case among 20,000 hospital admissions, 44 and it is the most common site of arteriovenous aneurysm because of the unique anatomy of an

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nociceptive responses at supraspinal sites could use lesions of the dorsolateral funiculus of the spinal cord to examine the possibility of direct supraspinal nociceptive suppression unrelated to changes in spinal cord input. Future studies in our laboratories will address these issues. Finally, it is important to note that the precise role of intralaminar nuclei in pain and temperature sensations has not been thoroughly characterized, and other thalamic regions may be more involved in pain perception. 4 We also found interesting Dr. Meyer's theory that hypothalamic

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René Tempelhoff, Paul A. Modica, Kerry L. Bernardo and Isaac Edwards

human limbic and thalamic regions. Anesthesiology 38 : 333 – 344 , 1973 Ferrer-Allado T, Brechner VL, Dymond A, et al: Ketamine-induced electroconvulsive phenomena in the human limbic and thalamic regions. Anesthesiology 38: 333–344, 1973 14. Frenk H : Pro- and anticonvulsant actions of morphine and the endogenous opioids: Involvement and interactions of multiple opiate and non-opiate systems. Brain Res 287 : 197 – 210 , 1983 Frenk H: Pro- and anticonvulsant actions of morphine and the endogenous opioids

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Yoichi Katayama, Takashi Tsubokawa, Tsuyoshi Maeda and Takamitsu Yamamoto

, depression transvelum interpositum * Voci, et al. , 34 and Russell and Rubinstein 25, 26 described one and two cases, respectively, without providing detailed information. Cases of cavernous malformations in the pineal, midbrain and thalamic regions or chiasma and optic nerve region are not detailed. — = no details available. † Their Case 21 appears to be the same case as that reported previously by Ishikawa, et al. , 8 and an additional case (Case 19) was reported without providing details. ‡ Their Case 2 was reported previously by

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Jamal M. Taha, Michele A. Janszen and Jacques Favre

, but refused to change the stimulation parameters that had achieved good tremor control. It is not clear whether this complication resulted from a bilateral microthalamotomy effect or from stimulation of both thalamic regions. Benabid, et al., 4 reported the absence of gross neuropsychological deficits after bilateral DBS; however, a detailed postoperative neuropsychological evaluation demonstrated a slight decline in verbal performance when the left Vim was stimulated, and in spatial performance when the right Vim was stimulated. We cannot comment on these findings