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Thomas H. Jones, Richard B. Morawetz, Robert M. Crowell, Frank W. Marcoux, Stuart J. FitzGibbon, Umberto DeGirolami and Robert G. Ojemann

paralysis threshold, function is reversibly lost; when flow drops further below an infarction threshold, anatomic damage becomes irreversible. Materials and Methods General Description Thirty-one Macaca irus monkeys of both sexes, weighing 3 to 5 kg, were studied. Under anesthesia, we installed an aortic catheter, intracerebral CBF electrodes, an intracranial pressure (ICP) transducer, and a snare ligature around the right MCA. After recovery from surgery, the MCA ligature was tightened for 15 minutes, 30 minutes, 2 to 3 hours, or permanently (see Table 3

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Jacob Rosenstein, Alexander Dah-Jium Wang, Lindsay Symon and Mikio Suzuki

/min, flattening of the EEG was noted. Thus, these thresholds appear very similar to those found in experimental animals. It has been suggested that ischemic thresholds for neuronal function may be somewhat higher in the unanesthetized animal. Jones, et al. , 17 defined a paralysis threshold of about 23 cc/100 gm/min above which no paralysis was noted in awake monkeys following MCA occlusion, and below which paralysis developed. The infarction threshold with permanent MCA occlusion in this model was noted at flows of about 17 to 18 cc/100 gm/min, and also appears to be