Search Results

You are looking at 1 - 10 of 48 items for :

  • "neurovascular compression syndrome" x
Clear All
Restricted access

Ghassan K. Bejjani and Laligam N. Sekhar

✓ Neurovascular compression syndromes are usually treated by interposing Teflon felt or padding or some other implant between the offending vessel and the nerves. However, this cannot be done in some cases in which ectatic vertebrobasilar arteries are involved. In these instances, alternative techniques must be used.

The authors report the use of a sling made of Prolene to reposition the vertebral artery in two patients with neurovascular compression disorder. The clinical results were gratifying, with complete resolution of the patients' symptoms.

Compression by large vessels is an uncommon but important source of neurovascular compression in patients with trigeminal neuralgia, hemifacial spasm, disabling positional vertigo, and, possibly, hypertension. The technique described may be useful to surgeons treating these problems.

Restricted access

Ricardo Segal, Peter J. Jannetta, Sidney K. Wolfson Jr., Manuel Dujovny and Eugene E. Cook

✓ A self-contained neurovascular compression simulator (NCS) has been designed to function as an artificial artery that pulsates with the heart. When implanted in animals, this device simulates those naturally occurring situations in which there is compression of nervous elements in the region of the brain stem or other areas by aberrant, or ectatic branches of normal arteries. The NCS consists of an intra-aortic balloon, a smaller (cephalic) balloon, a connecting tube, and an injection port, all fabricated of polyurethane-silicone compounds. With each heart systole, the rise in intra-aortic pressure is transmitted to the smaller cephalic balloon in the form of a pulsation. Thus, part of the cardiac ejection energy is transferred to the desired nervous structures. The performance of each NCS is tested in vitro in a pulse duplicator system.

The NCS was chronically implanted for up to 2 years in four dogs and 10 baboons. The cephalic balloon was placed intracranially in the subarachnoid space on the ventrolateral medulla adjacent to the entry zone of the ninth and 10th cranial nerves on the left side of baboons and on the right side of dogs. The position of the balloons was checked by fluoroscopy. Following implantation, the NCS could be inflated or deflated at will using the injection port which served to restart or discontinue the pulsations. No occlusion of the aorta or reduction of blood flow to the lower limbs or trunk was detected. By means of the NCS, an experimental model of neurogenic hypertension was produced in baboons.

Restricted access

Yu-Li Lee, Shu-Tian Chen, Jen-Tsung Yang, Hsu-Huei Weng, Hsueh-Lin Wang and Yuan-Hsiung Tsai

atrophy and demyelination in patients with TN. 29 However, not all patients with compressed nerves will be symptomatic, and the compression site of the nerves could be located anywhere along the cisternal segment. 2 The 3D MRI gradient echo sequence is an optimal tool for evaluating the degrees of TN that are caused by neurovascular compression syndrome (NVCS) and precisely indicates the affected range of the REZ. 2 , 10 , 24 The pretreatment severity of TN varies considerably in many patients who suffer from recurrent facial pain after medical and surgical

Restricted access

Takuro Inoue, Hisao Hirai, Toshiki Shimizu, Masayuki Tsuji, Ayako Shima, Fumio Suzuki and Masayuki Matsuda

, ephaptic neural transmission due to segmental demyelination stemming from a vascular problem may explain the possible pathogenesis of neurovascular compression syndrome. 18 , 19 This hypothesis seems to be widely accepted as an etiology in the seventh cranial nerve in hemifacial spasm, the fifth cranial nerve in trigeminal neuralgia, and, recently, the fourth cranial nerve in superior oblique myotonia. 14 , 15 Regarding the third cranial nerve, however, it is not uncommon in radiological findings that the PCA or SCA has contact with the nerve at the proximal cisternal

Restricted access

Makoto Oishi, Masafumi Fukuda, Tetsuya Hiraishi, Naoki Yajima, Yosuke Sato and Yukihiko Fujii

surgical procedures. 13 , 15 , 20 , 21 , 26 There are recent reports demonstrating a 3D interactive computer simulation system applicable not only to surveying 3D data visually but also to manipulating data by employing haptic sensations through a specific device. 13 , 15 , 26 Neurovascular compression syndromes, such as trigeminal neuralgia and hemifacial spasm, are defined as hyperactive dysfunctions involving the cranial nerves and have been explained as being caused by anatomical factors including vascular compression of the cranial nerve. 6 The success of MVD

Full access

Juergen Lutz, Niklas Thon, Robert Stahl, Nina Lummel, Joerg-Christian Tonn, Jennifer Linn and Jan-Hinnerk Mehrkens

integrity by measuring molecular diffusivity. 12 , 27 In a previous study, we showed that DTI—including the measurement of its main indices, namely fractional anisotropy (FA) and apparent diffusion coefficient (ADC)—enables the in vivo demonstration of microstructural changes within the affected trigeminal nerve in patients who suffer from a neurovascular compression syndrome. 27 In the current prospective study, we systematically evaluated FA and ADC values in a large patient cohort that underwent surgery for neurovascular compression-related TN and looked for possible

Restricted access

Daniel K. Resnick and Peter J. Jannetta

apparently iatrogenic cause of her symptoms following MVD of the seventh—eighth cranial nerve complex illustrates a rare complication of MVD, namely, iatrogenic cranial rhizopathy caused by shifting of arteries distant from the primary site of manipulation. Neurovascular compression syndromes have been implicated in the pathogenesis of multiple cranial rhizopathies. Strong clinical evidence supports the role of neurovascular compression in the pathogenesis of trigeminal neuralgia, hemifacial spasm, disabling vertigo, and glossopharyngeal neuralgia. Each of these disorders

Restricted access

Ramin Naraghi, Peter Hastreiter, Bernd Tomandl, Agatha Bonk, Walter Huk and Rudolf Fahlbusch

enough to describe or detect neurovascular compression. This is the main reason the results in several studies indicated no difference between patients with hypertension and normal individuals. 8, 19, 37, 38 Moreover, it is important to have information and an estimate about the course of the vessel and the relationships in the whole area ( Figs. 4 – 6 ). Interest in neurovascular relationships in the posterior fossa and the imaging of neurovascular compression syndromes is gradually increasing among radiologists. The knowledge of the neurosurgeon who regularly

Full access

Kazuhiko Nishino, Hitoshi Hasegawa, Kenichi Morita, Masafumi Fukuda, Yasushi Ito, Yukihiko Fujii and Mitsuya Sato

severe neurological deficits due to the initial hemorrhage. In addition, surgery might be indicated for AVMs with perinidal aneurysms unsuitable for embolization, causing repeat hemorrhages. On the other hand, elderly patients, who were frequently encountered in the present study, are likely to be candidates for GKS. As shown in Table 2 , CPAC AVMs presenting with neurovascular compression syndromes have been primarily managed by microsurgery; however, resection of the AVM frequently resulted in nerve root injury. 5 , 16 , 18 On this subject, preoperative MRI

Restricted access

Takashi Shimizu, Hiroto Kawasaki, Hidetoshi Kasuya and Koki Kurita

: Neurovascular compression syndromes , in Wilkins RH , Rengachary SS (eds): Neurosurgery. New York : Mc-Graw-Hill , 1985 , Vol 2 , pp 1901 – 1906 Jannetta PJ: Neurovascular compression syndromes, in Wilkins RH, Rengachary SS (eds): Neurosurgery. New York: Mc-Graw-Hill, 1985, Vol 2, pp 1901–1906 5. Keller PJ , Drayer BP , Fram EK , et al : MR angiography with two-dimensional acquisition and three-dimensional display. Work in progress. Radiology 173 : 527 – 532 , 1989 Keller PJ, Drayer BP