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Delayed traumatic intracerebral hematomas

Report of three cases

Frederick D. Brown, Sean Mullan, and Eugene E. Duda

W e have recently treated three patients with delayed traumatic intracerebral hematomas. The hematomas were insignificant or not present on initial computerized tomography (CT) scans. Repeat CT scans obtained following neurological deterioration of the patients revealed hematomas of significant mass that required surgical evacuation. There have been a few case reports of delayed intracerebral hemorrhage, 9, 11 and only one previous case occurred within the first 24 hours after injury as happened with two of our patients. 3 Case Reports Case 1 This

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Traumatic intracerebral hematoma

Report of 63 surgically treated cases

Kenneth G. Jamieson and John D. N. Yelland

. Intracerebral hematomas are often found beneath compound or simple depressed fractures; however, these have been excluded entirely from this series and will be reported separately later. They may also be found in relation to cerebral lacerations; these, having been considered in a previous report, are also excluded from this series. Six of the patients in this report also had extradural hematomas while 14 (including four with extradural hematomas) also had subdural hematomas; these 20 cases were therefore part of the previous reports as well as this one. In all 14 cases where

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Experimental intracerebral hematoma

Reduction of oxygen tension in brain and cerebrospinal fluid

Bernard J. Sussman, Jesse B. Barber, and Harold Goald

surgical removal. It is known that intracranial hemorrhage associated with subarachnoid bleeding in man induces a severe reduction of CSF oxygen tension that is not necessarily related to increased intracranial pressure. 24 This finding and the aforementioned considerations have led the authors to reevaluate intracerebral hematoma by experimental simulation in acute and short-term canine preparations. Method Experiment 1: Prolonged Measurement of Changes in the pH, pO 2 , pCO 2 of the Blood and CSF Under intravenous sodium pentobarbital anesthesia

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Chronic expanding intracerebral hematoma

Eugenio Pozzati, Giuliano Giuliani, Giulio Gaist, Giancarlo Piazza, and Gilberto Vergoni

intracerebral hematomas Case No. Age (yrs), Sex Symptoms Computerized Tomography Findings Surgical Findings * Outcome Density Ring Blush Edema Location 1 66, F headache, papilledema mixed yes yes parieto-occipital chronic encapsulated hematoma recurrent hematoma, dead 2 11, M seizures mixed yes yes frontoparietal chronic encapsulated hematoma good 3 16, F aphasia, rt hemiparesis hyperdense no yes temporal liquefied hematoma, AVM disabled 4 36, M

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Spontaneous intracerebral hematoma in carotid-cavernous fistula

Report of three cases

Donn M. Turner, John C. Vangilder, Saeid Mojtahedi, and Eric W. Pierson

H emorrhage associated with carotid-cavernous fistula (CCF) has been reported to occur in 3% of patients. 12, 34, 44 Although CCF has long been recognized as a threat to vision, only a few authors have documented the danger of spontaneous rupture of the fistula. 9, 11, 33, 34, 39, 44 In this paper, we report three cases of spontaneous intracerebral hematoma in patients with CCF's. In each case, the hemorrhage occurred in the vicinity of localized venous engorgement, as demonstrated by cerebral angiography. A mechanism for the bleeding is proposed, and the

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Encapsulated intracerebral hematomas: a defined entity

Case report

José M. Roda, Fernando Carceller, Antonio Pérez-Higueras, and Carmen Morales

: Computed tomography in intracerebral hematomas. I. Transmission CT observations on hematoma resolution. AJR 129: 681–688, 1977 6. Fiumara E , Gambacorta M , D'Angelo V , et al : Chronic encapsulated intracerebral haematoma: pathogenetic and diagnostic considerations. J Neurol Neurosurg Psychiatry 52 : 1296 – 1299 , 1989 Fiumara E, Gambacorta M, D'Angelo V, et al: Chronic encapsulated intracerebral haematoma: pathogenetic and diagnostic considerations. J Neurol Neurosurg Psychiatry 52: 1296–1299, 1989 7

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Stereotaxic evacuation of spontaneous intracerebral hematomas

Edward I. Kandel and Vjacheslaw V. Peresedov

I n 1978, Backlund and von Holst 1 proposed a new principle of stereotaxic subtotal removal of intracerebral hematomas in which a special instrument with a mandrel-like Archimedes screw inside a cannula is used to destroy and remove dense clots. We have improved on this promising method and have designed instrumentation for use in this operative procedure. In this paper, we describe the device and its use in 32 cases of spontaneous intracerebral hematoma, including one case caused by aneurysmal rupture and one secondary to rupture of an arteriovenous

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Intracerebral Hematoma and Aneurysms

Neurosurgical Forum: Letters to the Editor To The Editor Dimitris Antoniou , M.D. Iaso General Hospital Athens, Greece 582 582 Abstract Object. The aim of this study was to evaluate the efficacy of a treatment combination of coil embolization and clot evacuation in patients presenting with an intracerebral hematoma (ICH) caused by the rupture of an aneurysm. Methods. Twenty-seven patients were prospectively recruited in this study between 1996 and 2000. Endovascular treatment of the putative ruptured

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Tentorial arteriovenous malformation presenting as an intracerebral hematoma

Case report

Raeph Fardoun, Yves Adam, Philippe Mercier, and Gilles Guy

D ural arteriovenous malformations (AVM's) occur infrequently. Although the Cooperative Study did not mention any instances, Newton, et al. , 5, 6 found 15 cases in a series of 129 patients with angiomas. We would like to report an unusual case of a patient who presented with intracerebral hematoma in a purely dural fistula, and to discuss its physiopathology. Case Report This 54-year-old cabinet-maker was hospitalized for evaluation of the sudden onset of a headache while straining. He had a 5-year history of hypertension. On examination, he was

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CT-guided stereotaxic evacuation of hypertensive intracerebral hematomas

Keizo Matsumoto and Hideki Hondo

hematoma was 3 days or less (acute stage) in 37 cases, between 4 and 14 days (subacute stage) in nine cases, and 15 days or more (chronic stage) in five cases. The hemorrhage was in the basal ganglion in 34 patients, and was subcortical in 11, thalamic in three, and cerebellar in three ( Table 1 ). TABLE 1 Location and stage of hypertensive intracerebral hematomas evacuated in 51 cases Location of Hematoma Stage Total Cases Acute Subacute Chronic basal ganglia 27 5 2 34 subcortex 4 4 3 11