Search Results

You are looking at 1 - 10 of 426 items for :

  • "hemifacial spasm" x
Clear All
Restricted access

Hemifacial spasm

Comparison of three different operative procedures in 110 patients

Tsutomu Iwakuma, Akihisa Matsumoto and Nishio Nakamura

different operative procedures for treating hemifacial spasm: partial sectioning of the seventh nerve just distal to the stylomastoid foramen; selective facial neurectomy; and microvascular decompression of the facial nerve in the cerebellopontine angle. This paper compares these methods. Neuropathological and electromyographic findings are described and the mechanism of synkinesis is discussed. Clinical Material This series includes 110 patients (33 men and 77 women) aged 18 to 75 years, who presented with persistent intractable hemifacial spasm and no previous

Restricted access

A. Peter Moore

T heories of the etiology of hemifacial spasm range from a central or brain-stem mechanism 1 to either intracranial 2 or extracranial 5 lesions of the facial nerve. The case is reported of a patient who harbored a large fusiform aneurysm impinging on the facial nerve, causing fluctuating hemifacial spasm. Case Report This 52-year-old man had been cut on the back of his head by a piece of coal in a mining accident 6 years before his present admission. He did not lose consciousness. About 1 month later, he noted twitching of his right eyelid when his

Restricted access

Fred G. Barker II, Peter J. Jannetta, David J. Bissonette, Philip T. Shields, Mark V. Larkins and Hae Dong Jho

H emifacial spasm is an uncommon disorder characterized by involuntary paroxysmal movement of one side of the face. Since the 1960s 13, 14 there has been increasing consensus that this disorder may be relieved by repositioning of arteries in contact with the facial nerve in its course through the subarachnoid cisterns of the posterior fossa. 23, 26, 41 This report describes our experience with microvascular decompression for hemifacial spasm in 703 patients (705 sides) over a 20-year period. Clinical Material and Methods Patient Population All

Restricted access

Microvascular decompression for hemifacial spasm

Patterns of vascular compression in unsuccessfully operated patients

Shinji Nagahiro, Akira Takada, Yasuhiko Matsukado and Yukitaka Ushio

M icrovascular decompression via retromastoid craniectomy is the preferred method for treating hemifacial spasm. 1, 2, 4–8, 10, 11 However, treatment failure, failure to relieve the spasm, and recurrence of the spasm have been reported. 1, 2, 3–8, 10, 11 Little work has been carried out to determine the factors causing unsuccessful microvascular decompression. We examined the causative factors in patients who had undergone unsuccessful microvascular decompression in a retrospective study of the relationship between the follow-up results and the pattern of

Restricted access

Han-Jung Chen, Tao-Chen Lee and Chun-Chung Lui

disappeared 1 week after surgery without any neurological deficit. Discussion Hemifacial spasm caused by venous compression is not rare. However, a case in which the offending vessel is a result of a venous angioma is extremely rare. 5 Cerebral angiography in this case revealed the fact that a large area of the cerebellar venous drainage depended on this angioma. Preservation of the vein is thus a key point for treatment of hemifacial spasm and prevention of any possibility of venous infarction. 1, 4 References 1. Huang YP , Robbins A

Restricted access

Douglas C. Bills and Ahmed Hanieh

T he first reported case of hemifacial spasm was described in 1875 by Schultze; 22 in that case, an aneurysm of the vertebral artery was seen at autopsy to be compressing the facial nerve at its exit from the brain stem. Further sporadic reports followed, apparently caused by a variety of compressing lesions. Still, the majority of cases were classified as idiopathic, and treatments were largely empirical. The theory of microvascular compression by aberrant arterial or venous loops, as championed by Jannetta, et al. , 7 has more recently been accepted as an

Restricted access

Mauricio Campos-Benitez and Anthony M. Kaufmann

aneurysm clips in microvascular decompression surgery. Technical note and case series . J Neurosurg 106 : 929 – 931 , 2007 2 Barker FG II , Jannetta PJ , Bisonette DJ , Shields PT , Larkins MV , Jho HD : Microvascular decompression for hemifacial spasm . J Neurosurg 82 : 201 – 210 , 1995 3 De Ridder D , Moller A , Verlooy J , Cornelissen M , De Ridder Leo : Is the root entry/exit zone important in microvascular compression syndromes? . Neurosurgery 51 : 427 – 433 , 2002 4 Gardner WJ : Concerning the mechanism of

Restricted access

Margareta B. Møller and Aage R. Møller

C lassical hemifacial spasm is a disorder characterized by a hyperactive dysfunction of the facial nerve leading to progressive involuntary twitching of the muscles of the face. The spasm is most often unilateral, occurs more commonly in women than in men, and affects the left side more often than the right. There is now extensive evidence that the spasm is usually caused by cross compression of the seventh cranial nerve at its root entry zone, 6, 7, 9 and a study of a large series of patients has shown that microvascular decompression of the seventh

Restricted access

Observations on synkinesis in patients with hemifacial spasm

Effect of microvascular decompression and etiological considerations

Phyo Kim and Takanori Fukushima

H emifacial spasm is a distressing movement disorder of the face characterized by paroxysmal involuntary contractions of muscles innervated by the unilateral facial nerve. The primary etiology is unknown, and various medical therapies or surgical procedures disrupting facial nerve conduction have failed to yield satisfactory results. Gardner and Sava 7 first suggested vascular compression of the intracranial portion of the facial nerve as the cause of hemifacial spasm; however, this concept was not widely accepted. Jannetta, et al. , 11 have clearly

Restricted access

Etiology and definitive microsurgical treatment of hemifacial spasm

Operative techniques and results in 47 patients

Peter J. Jannetta, Munir Abbasy, Joseph C. Maroon, Francisco M. Ramos and Maurice S. Albin

. Pain is not a prominent symptom, although patients with the tonus phenomenon may develop an aching discomfort. The patients have no symptoms of other cranial nerve dysfunction. The problem is often confused with a nervous habit spasm or “tic,” and patients are frequently sent for psychiatric help. Hemifacial spasm may become socially, psychologically, and frequently economically disabling. Treatment has generally consisted of the application of mild to severe trauma to the facial nerve. The authors have had an opportunity to evaluate and treat 45 patients with