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Kewal K. Jain

mm craniotomy, plastic coating of aneurysm, 1973 no relief of headaches TABLE 2 Results in three nontreated patients Case No. Age, Sex Clinical Features Aneurysm Location, Size Follow-up 13 50 F headaches, alcoholism grand mal seizures lt middle cerebral, 1.5 cm; diagnosed 1970 still alive, no change in clinical status 14 69 F sudden onset lt headache with lt pupil larger than rt, hypertension bil post communicating, lt 5 mm, rt 3 mm; diagnosed 1969 no recurrence of headache

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Sharon Rivas, G. Logan Douds, Roger H. Ostdahl and Kimberly S. Harbaugh

other clinically relevant antiganglioside antibodies in these patients are ongoing. 40 From a practical standpoint, antiganglioside antibody testing is not helpful in the immediate treatment of these patients because test results are not available until several days after the samples are obtained. 24 Differential Diagnosis Given the patient's history of alcoholism and hyponatremia, the differential diagnosis includes central pontine myelinolysis. 22 , 32 However, his neurological decline occurred when he was mildly hyponatremic and not following normalization

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Paul E. Kaloostian, Han Chen, Frederick Rupp and Erich Marchand

report . Turk Neurosurg 17 : 224 – 227 , 2007 3 Giuffrè R : Physiopathogenesis of chronic subdural hematomas: a new look to an old problem . Riv Neurol 57 : 298 – 304 , 1987 4 Gjerris F , Schmidt K : Chronic subdural hematoma: surgery or mannitol treatment . J Neurosurg 40 : 639 – 642 , 1974 5 Hadjigeorgiou G , Chamilos C , Petsanas A , Vranos G , Foteas P , Spiridakis F : Rapid spontaneous resolution of acute subdural hematoma in a patient with chronic alcoholism . Br J Neurosurg 26 : 415 – 416 , 2012 6 Kawano N

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found a significant relation between epidural empyema after bone flap reimplantation and the presence of a VP shunt at the time of cranial vault reconstruction, the implantation of a VP shunt together (p=0.001) with the bone flap and the implantation of fragmented bone flaps (p=0.016). We also found a significant relation between aseptic necrosis and an open traumatic brain injury (open TBI) as primary diagnosis (p=0.004). Further alcoholism was identified as a risk factor for aseptic bone flap necrosis (p=0.010). Conclusions In order to avoid infectious

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Craig J. McClain, Diana L. Twyman, Linda G. Ott, Robert P. Rapp, Phillip A. Tibbs, Jane A. Norton, Edward J. Kasarskis, Robert J. Dempsey and Byron Young

night blindness and hypogonadism. Alcoholism Clin Exp Res 3 : 135 – 141 , 1979 McClain CJ, Van Thiel DH, Parker S, et al: Alterations in zinc, vitamin A, and retinol-binding protein in chronic alcoholics: a possible mechanism for night blindness and hypogonadism. Alcoholism Clin Exp Res 3: 135–141, 1979 45. Miller SL : The metabolic response to head injury. S Afr Med J 65 : 90 – 91 , 1984 Miller SL: The metabolic response to head injury. S Afr Med J 65: 90–91, 1984 46. Pories WJ , Henzel JH

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Abdul Rasul Sadik, Masazumi Adachi and Joseph Ransohoff

hemiparesis cleared rapidly. Marked improvement in his organic mental changes was noted. Roentgenogram of the skull showed the clips in contact. An electroencephalogram on July 28, 1962 was reported as normal. Psychiatric evaluation was that of a mild organic mental syndrome secondary to chronic alcoholism and head trauma. He was discharged from the hospital on Aug. 8, 1962, ambulatory, and was fully rational with no focal neurological deficit. Discussion Clark and Gooddy 2 classified ruptured intracranial aneurysm as (1) rupture resulting in extensive

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Shashi Dhawan, Deepali Jain and Veer Singh Mehta

bacteria. 10 Human infection is usually associated with intestinal symptoms, such as diarrhea and dysentery, but ranges from an asymptomatic carrier state through a chronic symptomatic infection presenting with nonbloody diarrhea to a dysentery-like picture. Balantidium coli can thrive in the colon in balance with its host without causing dysenteric symptoms, but malnutrition, alcoholism, or a compromised immune system can tip the balance in favor of the ciliate, leading to disease. 2 , 18 Our patient belonged to a family of farmers and was a known alcoholic with

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Avital Fast, Malvina Alon, Shmuel Weiss and Freddy R. Zer-Aviv

changes in pharmacotherapy. Acta Orthop Scand 53: 853–856, 1982 9. Habermann ET , Cristofaro RL : Avascular necrosis of bone as a complication of renal transplantation. Semin Arthritis Rheum 6 : 189 – 206 , 1976 Habermann ET, Cristofaro RL: Avascular necrosis of bone as a complication of renal transplantation. Semin Arthritis Rheum 6: 189–206, 1976 10. Hungerford DS , Zizic TM : Alcoholism associated ischemic necrosis of the femoral head. Early diagnosis and treatment. Clin Orthop 130 : 144

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not advocate aggressive management of hyponatremia and hypochloremia. We suggested that replacement of Na and Cl loss begin in the operating room, after the need for brain relaxation has ceased. This is accomplished by the judicious use of 0.45% NaCl in D5W infusion, with repeated measurements of plasma Na and Cl. The claim that rapid correction of hyponatremia may lead to central pontine myelinolysis (CPM) is not pertinent here. We currently believe that CPM is a rare puzzling syndrome characterized by the triad of alcoholism and prolonged hyponatremia (lasting

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Luis E. Savastano, Todd C. Hollon, Ariel L. Barkan and Stephen E. Sullivan

K orsakoff syndrome is a neuropsychiatric disorder characterized by severe memory deficits generally disproportionate to any other impairment in cognitive function. 9 , 23 Since the original description of this condition by Sergei Korsakoff in 1887, the syndrome has been associated with thiamine deficiency in the setting of chronic alcoholism or other disease related to severe malnutrition or malabsorption, such as prolonged intravenous feeding, cancer, or acquired immunodeficiency syndrome. 4 , 18 However, some have reported that focal structural lesions