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James K. Liu, Michael S. Tenner, Oren N. Gottfried, Edwin A. Stevens, Joshua M. Rosenow, Neel Madan, Joel D. Macdonald, John R. W. Kestle and William T. Couldwell

C erebral vasospasm caused by aneurysmal SAH continues to be a disabling disease associated with high risks of morbidity and death. Furthermore, it is the most common cause of delayed neurological deficits, affecting up to 30% of patients who survive the initial hemorrhage. 17, 23 Current therapy for prevention and treatment of symptomatic vasospasm includes triple-H therapy, 1 Ca channel antagonists, 2 and early surgery with clot removal. 15 If, however, vasospasm is refractory to medical management, endovascular therapy, consisting of selective