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Intratumoral abscess complicating a postradiation-induced World Health Organization grade II meningioma: illustrative case

Katherine Callahan, Isidora Beach, Sadie Casale, John DeWitt, and Bruce Tranmer

Meningiomas are the most commonly diagnosed benign primary brain mass, accounting for more than 35% of all brain neoplasms diagnosed annually. 1 In the Western world, brain abscesses occur at a rate of approximately 4 per 1 million annually and are most commonly due to infection by Staphylococcus or Streptococcus bacteria. 2 Peritumoral abscesses are historically associated with intra- or parasellar tumors and have predominantly originated from direct contact with the prenasal sinuses. 3 However, in most reported cases of meningioma-associated abscess

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Strongyloides hyperinfection syndrome due to corticosteroid therapy after resection of meningioma: illustrative case

Víctor Rodríguez Domínguez, Carlos Pérez-López, Catalina Vivancos Sánchez, Cristina Utrilla Contreras, Alberto Isla Guerrero, and María José Abenza Abildúa

corticosteroids in neurosurgery (e.g., chronic subdural hematoma, brain tumor, inflammatory pain, hypopituitarism) for long periods (before surgery, during the perioperative period, and after the surgery), it is important in neurosurgical practice to know the risk factors, epidemiology, and management to avoid delays in diagnosis and prevent the high mortality that this disease entails. 4 , 5 We report a case of Strongyloides hyperinfection syndrome in a patient diagnosed with a large sphenoid planum meningioma and treated with corticosteroids. Illustrative Case A

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Intracranial aspergilloma mimicking metachronous meningioma following transsphenoidal removal of a tuberculum sellae meningioma: illustrative case

Daisuke Sato, Hirotaka Hasegawa, Hironobu Nishijima, Kyotaro Kawase, Koh Okamoto, Akiko Iwasaki, Yuki Shinya, Masahiro Abe, Yoshitsugu Miyazaki, and Nobuhito Saito

complication following eTSS. Post-eTSS CNS aspergillosis infection has been reported infrequently, usually presenting as a mycotic aneurysm subsequent to vasculitis or a local fungus ball formation. 7–9 Here, we describe a case of CNS aspergilloma that developed over 6 months following eTSS for a tuberculum sellae meningioma in an immunocompetent patient who had concurrent asymptomatic fungal sinusitis at the time of the initial eTSS. Illustrative Case A 58-year-old woman was referred to our department following incidental detection of a tuberculum sellae meningioma

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Intracranial tuberculoma: a rare complication of extrapulmonary tuberculosis. Illustrative case

Vijay Letchuman, Andrew R. Guillotte, Paige A. Lundy, Anand Dharia, Nelli S. Lakis, and Paul J. Camarata

findings, it could have represented a superficial primary glial neoplasm such as a ganglioglioma or pleomorphic xanthoastrocytoma. Along with gliomas, the differential diagnosis includes meningioma, intracranial abscess, cerebral metastases, and other CNS infectious etiologies. 12 The importance of definitive diagnosis was urgently pursued in this case due to the interval progression of radiographic features along with the persistence of breakthrough seizures while on consistent first-line antiepileptic therapy. Other indications for resection of these masses includes

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Presentation, diagnosis, and treatment of a cerebellar tuberculoma: illustrative case

Stephen Capone, Dokpe Emechebe, Eric G. St. Clair, Ali Sadr, and Michelle Feinberg

Kings County Hospital Center emergency department in Brooklyn, New York, with a 3-month history of severe right temporo-occipital headaches with photophobia and night sweats. The patient denied fevers, cough, chest pain, shortness of breath, and weight loss. On presentation, the patient was neurologically intact and afebrile, and laboratory studies were unremarkable ( Table 1 ). A head CT scan showed a partially calcified 2.5 × 2.0 cm extra-axial right cerebellar mass with adjacent vasogenic edema ( Fig. 1 ). The differential included a calcified meningioma versus an

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Coccidioidal meningitis with multiple aneurysms presenting with pseudo–subarachnoid hemorrhage: illustrative case

Rohin Singh, Visish M. Srinivasan, Joshua S. Catapano, Joseph D. DiDomenico, Jacob F. Baranoski, and Michael T. Lawton

, Alvarez-Vega MA , Gutierrez-Morales JC , Lopez-Garcia A . Meningioma associated with brain aneurysm: report of two cases . Turk Neurosurg . 2017 ; 27 ( 2 ): 321 – 323 . 32053327 12 Signorelli F , Sela S , Gesualdo L , Hemodynamic stress, inflammation, and intracranial aneurysm development and rupture: a systematic review . World Neurosurg . 2018 ; 115 : 234 – 244 . 10.1016/j.wneu.2018.04.143 31597500 13 Lee H , Kim T . Rapid development of intracranial aneurysm associated with tuberculous meningitis . Can J Neurol Sci

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Wound vacuum-assisted closure as a bridge therapy in the treatment of infected cranial gunshot wound in a pediatric patient: illustrative case

Harjus Birk, Audrey Demand, Sandeep Kandregula, Christina Notarianni, Andrew Meram, and Jennifer Kosty

Powers et al., 2013 5 69/M Invasive scalp squamous cell carcinoma Infection 91 Latissimus free flap Death due to respiratory failure 73/M Subdural empyema Infection 30 Family transitioned to comfort care Death due to respiratory failure 75/F Invasive atypical meningioma Infection 71 STSG Good wound healing 73/M Invasive sinonasal carcinoma Infection 16 Family transitioned to comfort care Death due to sepsis 24/M Cranial gunshot wound Infection 32 Delayed primary closure Good wound healing

Open access

Trigeminal neuralgia secondary to vascular compression and neurocysticercosis: illustrative case

Mao Vásquez, Luis J. Saavedra, Hector H. García, Evelyn Vela, Jorge E. Medina, Miguel Lozano, Carlos Hoyos, and William W. Lines-Aguilar

tumors (meningiomas, epidermoid cysts, acoustic neuromas, etc.) and, less frequently, aneurysms and arteriovenous malformations, 5 and rarer still, neurocysticercosis (NCC). In secondary TN, pathophysiological changes similar to those in primary TN occur, although the structural lesion depends on the etiology; for example, in multiple sclerosis, it is due to demyelination plaques. 7 The standard treatment for TN is microsurgical decompression of the trigeminal nerve. We present a case in which neighboring NCC cysts, arachnoiditis, and vascular compression occurred in