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Syringomyelia intermittens: highlighting the complex pathophysiology of syringomyelia. Illustrative case

Jorn Van Der Veken, Marguerite Harding, Saba Hatami, Marc Agzarian, and Nick Vrodos

intracranial pressure due to a space-occupying lesion (chronic subdural hematomas, cavernoma), hydrocephalus, head injuries, and venoocclusive disease have been described as a cause for tonsillar herniation. None of these was present in our patient. 40–44 A CSF leak as a cause of reduced intracranial pressure and subsequent resolution of CMI has also been reported. 28 Other pathophysiological mechanisms for spontaneous resolution have been proposed, when a change in tonsillar descent is absent or less clear. In 1991, Jack et al. published a hypothesis of spontaneous

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Pathophysiology and surgical treatment of spinal adhesive arachnoid pathology: patient series

Izumi Koyanagi, Yasuhiro Chiba, Genki Uemori, Hiroyuki Imamura, Masami Yoshino, and Toshimitsu Aida

hemorrhage from ruptured basilar aneurysm or dissection of the vertebral artery. The other patient suffered from acute spinal subdural hematoma of unknown origin. Trauma as the etiology was noted in 2 patients (severe head injury in 1; spinal cord injury in 1). Another 4 patients had different types of etiology: tuberculosis (caries), pyogenic meningitis, oil-myelography, and cerebrospinal fluid (CSF) leakage syndrome. The interval from etiological events to onset of symptoms of arachnoid adhesion in these 13 patients ranged from 1 to 744 months (mean 167.3 months, median