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Michael F. Stiefel, Alejandro Spiotta, Vincent H. Gracias, Alicia M. Garuffe, Oscar Guillamondegui, Eileen Maloney-Wilensky, Stephanie Bloom, M. Sean Grady, and Peter D. LeRoux

CPP and brain O 2 is not observed in every patient, in large part because autoregulation frequently is disturbed. 39 Results from recent studies have also demonstrated that an increase in brain tissue PO 2 is associated with improved cerebral metabolism. 32, 45 Together these data indicate that it is reasonable to assume that the use of a brain tissue PO 2 monitor and efforts to increase brain O 2 delivery may improve TBI outcome. Brain Tissue PO 2 —Guided Therapy and Outcome Results of this study demonstrate that brain tissue PO 2 —guided treatment is

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Alejandro M. Spiotta, Michael F. Stiefel, Vicente H. Gracias, Alicia M. Garuffe, W. Andrew Kofke, Eileen Maloney-Wilensky, Andrea B. Troxel, Joshua M. Levine, and Peter D. Le Roux

randomised controlled trial . Lancet Neurol 6 : 29 – 38 , 2007 53 Tolias CM , Reinert M , Seiler R , Gilman C , Scharf A , Bullock MR : Normobaric hyperoxia—induced improvement in cerebral metabolism and reduction in intracranial pressure in patients with severe head injury: a prospective historical cohort-matched study . J Neurosurg 101 : 435 – 444 , 2004 54 Valadka AB , Gopinath SP , Contant CF , Uzura M , Robertson CS : Relationship of brain tissue PO2 to outcome after severe head injury . Crit Care Med 26 : 1576 – 1581